Besides selenium, I have not eaten in a restaurant or anyones home for 15 yrs-I have only eaten my own macrobiotic food, all of which are SUPERFOODS–KNOCK ON WOOD, MY IMMUNE SYSTEM IS HIGHLY FUNCTIONING-I have not had a cold or flu for 25 yrs including living with my wife and son who had colds and flu–
I suggest that all of you bookmark this link to a site entitled “The Worlds Healthiest Foods” –ALL the info is from peer review journals…if u want good heath…if u want immunity…you have to CHANGE YOUR LIFE STYLE and get off your butts and learn what real foods are…if not, that’s fine too…my motto is live and let live…so go ahead and take your chances and keep eating from the carcinogenic menu from
DINERS, DRIVE INS AND DIVES—–[one of my favorite shows…seriously]
http://www.whfoods.com/foodstoc.php
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Lipinski 2015, Ebola and Selenium: How not to catch the 2019 Novel Coronavirus (2019-nCoV)
The only people that can be infected by the 2019-n Coronavirus have less than 98.7 µg/L of
Selenium in plasma or serum. Those who have enough Selenium are immune to this and all other enveloped viruses. Selenium can be obtained from Brazil nuts, Selenium pills or Astragalus tea.
This is why only some people get the flu and why others get it infrequently or never at all.
We only found this out in 2015 when Lipinski @ Harvard figured out why some people were immune to Ebola, a fact well documented in medical archives.
Two Brazil nuts a day will do it. An Asia astragalus tea is the primary source of selenium.
2015 Lipinski – Can Selenite Be An Ultimate Inhibitor Of Ebola And Other Viral Infections?
“It is known that the virulence of Ebola and other RNA enveloped viruses involves in the first step their attachment to host cell membranes. Following this initial step the virus enters the target cell cytoplasm by forming hydrophobic spikes that make holes in the membrane lipid bilayer. Formation of such spikes is catalyzed by the reduced form of viral protein disulfide isomerase (PDIred) thus initiating chain of disulfide exchange reactions. Consequently, hydrophobic protein epitopes become exposed, which in the absence of proper chaperones form hydrophobic ‘spikes’ capable of penetrating the host cell membranes.